Relaxin Suppresses Atrial Fibrillation by Reversing Fibrosis and Myocyte Hypertrophy and Increasing Conduction Velocity and Sodium Current in Spontaneously Hypertensive Rat Hearts | Zendy

Ashish Parikh | Zendy; Divyang Patel | Zendy; Charles F. McTiernan | Zendy; Wenyu Xiang | Zendy; Jamie Haney | Zendy; Lei Yang | Zendy; Bo Lin | Zendy; Aaron D. Kaplan | Zendy; Glenna C.L. Bett | Zendy; Randall L. Rasmusson | Zendy; Sanjeev G. Shroff | Zendy; David Schwartzman | Zendy; Guy Salama | Zendy

Open Access

Relaxin Suppresses Atrial Fibrillation by Reversing Fibrosis and Myocyte Hypertrophy and Increasing Conduction Velocity and Sodium Current in Spontaneously Hypertensive Rat Hearts

Author(s) -

Ashish Parikh,

Divyang Patel,

Charles F. McTiernan,

Wenyu Xiang,

Jamie Haney,

Lei Yang,

Bo Lin,

Aaron D. Kaplan,

Glenna C.L. Bett,

Randall L. Rasmusson,

Sanjeev G. Shroff,

David Schwartzman,

Guy Salama

Publication year - 2013

Publication title -

circulation research

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 4.899

H-Index - 336

eISSN - 1524-4571

pISSN - 0009-7330

DOI - 10.1161/circresaha.113.301646

Subject(s) - medicine , endocrinology , fibrosis , nerve conduction velocity , relaxin , muscle hypertrophy , myocardial fibrosis , hormone

Atrial fibrillation (AF) contributes significantly to morbidity and mortality in elderly and hypertensive patients and has been correlated to enhanced atrial fibrosis. Despite a lack of direct evidence that fibrosis causes AF, reversal of fibrosis is considered a plausible therapy.

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