Genetic Deletion of Chemokine Receptor Ccr6 Decreases Atherogenesis in  ApoE  -Deficient Mice | Zendy

Wuzhou Wan | Zendy; Jean K. Lim | Zendy; Michail S. Lionakis | Zendy; Aymeric Rivollier | Zendy; David H. McDermott | Zendy; Brian L. Kelsall | Zendy; Joshua Μ. Farber | Zendy; Philip M. Murphy | Zendy

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Genetic Deletion of Chemokine Receptor Ccr6 Decreases Atherogenesis in ApoE -Deficient Mice

Author(s) -

Wuzhou Wan,

Jean K. Lim,

Michail S. Lionakis,

Aymeric Rivollier,

David H. McDermott,

Brian L. Kelsall,

Joshua Μ. Farber,

Philip M. Murphy

Publication year - 2011

Publication title -

circulation research

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 4.899

H-Index - 336

eISSN - 1524-4571

pISSN - 0009-7330

DOI - 10.1161/circresaha.111.242578

Subject(s) - c c chemokine receptor type 6 , chemokine receptor , chemokine , apolipoprotein e , immunology , biology , cc chemokine receptors , receptor , chemokine receptor ccr5 , medicine , genetics , inflammation , disease

The chemokine receptor Ccr6 is a G-protein-coupled receptor expressed on various types of leukocytes identified in mouse atherosclerotic lesions. Recent evidence suggests that both CCR6 and its ligand CCL20 are also present in human atheroma; however, their functional roles in atherogenesis remain undefined.

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