Intravenous Gene Therapy With PIM-1 Via a Cardiotropic Viral Vector Halts the Progression of Diabetic Cardiomyopathy Through Promotion of Prosurvival Signaling | Zendy

Rajesh Katare | Zendy; Andrea Caporali | Zendy; Lorena Zentilin | Zendy; Elisa Avolio | Zendy; Graciela Sala-Newby | Zendy; Atsuhiko Oikawa | Zendy; Daniela Cesselli | Zendy; Antonio Paolo Beltrami | Zendy; Mauro Giacca | Zendy; Costanza Emanueli | Zendy; Paolo Madeddu | Zendy

Open Access

Intravenous Gene Therapy With PIM-1 Via a Cardiotropic Viral Vector Halts the Progression of Diabetic Cardiomyopathy Through Promotion of Prosurvival Signaling

Author(s) -

Rajesh Katare,

Andrea Caporali,

Lorena Zentilin,

Elisa Avolio,

Graciela Sala-Newby,

Atsuhiko Oikawa,

Daniela Cesselli,

Antonio Paolo Beltrami,

Mauro Giacca,

Costanza Emanueli,

Paolo Madeddu

Publication year - 2011

Publication title -

circulation research

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 4.899

H-Index - 336

eISSN - 1524-4571

pISSN - 0009-7330

DOI - 10.1161/circresaha.110.239111

Subject(s) - diabetic cardiomyopathy , cancer research , cardiac fibrosis , apoptosis , biology , cardiac function curve , cardiomyopathy , progenitor cell , downregulation and upregulation , heart failure , medicine , endocrinology , microbiology and biotechnology , stem cell , biochemistry , gene

Studies in transgenic mice showed the key role of (Pim-1) (proviral integration site for Moloney murine leukemia virus-1) in the control of cardiomyocyte function and viability.

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