CD36 Is a Matrix Metalloproteinase-9 Substrate That Stimulates Neutrophil Apoptosis and Removal During Cardiac Remodeling | Zendy

Kristine Y. DeLeonPennell | Zendy; Yuan Tian | Zendy; Bai Zhang | Zendy; Courtney Cates | Zendy; Rugmani Padmanabhan Iyer | Zendy; Presley L. Can | Zendy; Punit Shah | Zendy; Paul Aiyetan | Zendy; Ganesh V. Halade | Zendy; Yonggang Ma | Zendy; Elizabeth O’Flynn | Zendy; Zhen Zhang | Zendy; Yu-Fang Jin | Zendy; Hui Zhang | Zendy; Merry L. Lindsey | Zendy

Open Access

CD36 Is a Matrix Metalloproteinase-9 Substrate That Stimulates Neutrophil Apoptosis and Removal During Cardiac Remodeling

Author(s) -

Kristine Y. DeLeonPennell,

Yuan Tian,

Bai Zhang,

Courtney Cates,

Rugmani Padmanabhan Iyer,

Presley L. Can,

Punit Shah,

Paul Aiyetan,

Ganesh V. Halade,

Yonggang Ma,

Elizabeth O’Flynn,

Zhen Zhang,

Yu-Fang Jin,

Hui Zhang,

Merry L. Lindsey

Publication year - 2015

Publication title -

circulation cardiovascular genetics

Language(s) - English

Resource type - Journals

eISSN - 1942-325X

pISSN - 1942-3268

DOI - 10.1161/circgenetics.115.001249

Subject(s) - cd36 , matrix metalloproteinase , apoptosis , biology , phagocytosis , downregulation and upregulation , medicine , immunology , microbiology and biotechnology , endocrinology , biochemistry , receptor , gene

After myocardial infarction, the left ventricle undergoes a wound healing response that includes the robust infiltration of neutrophils and macrophages to facilitate removal of dead myocytes as well as turnover of the extracellular matrix. Matrix metalloproteinase (MMP)-9 is a key enzyme that regulates post-myocardial infarction left ventricular remodeling.

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