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Lack of Uniform Progression of Endocardial Scar in Patients With Arrhythmogenic Right Ventricular Dysplasia/Cardiomyopathy and Ventricular Tachycardia
Author(s) -
Michael P. Riley,
Erica S. Zado,
Rupa Bala,
David J. Callans,
Joshua M. Cooper,
Sanjay Dixit,
Fermin C. García,
Edward P. Gerstenfeld,
Mathew D. Hutchinson,
David Lin,
Vickas Patel,
Ralph J. Verdino,
Francis E. Marchlinski
Publication year - 2010
Publication title -
circulation arrhythmia and electrophysiology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.684
H-Index - 102
eISSN - 1941-3149
pISSN - 1941-3084
DOI - 10.1161/circep.109.919530
Subject(s) - medicine , arrhythmogenic right ventricular dysplasia , cardiology , ventricular tachycardia , ablation , sinus rhythm , cardiomyopathy , endocardium , ventricle , population , tachycardia , dilated cardiomyopathy , heart failure , atrial fibrillation , environmental health
The endocardial substrate for ventricular arrhythmias in patients with arrhythmogenic right ventricular dysplasia/cardiomyopathy (ARVD/C) is thought to be caused by a progressive degenerative process. Many clinical decisions and treatment plans are guided by this pathophysiologic assumption, but the extent of progression of macroscopic endocardial scar and right ventricular (RV) dilatation have not been assessed.

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