Lipoprotein(a) and HIV
Author(s) -
Byambaa Enkhmaa,
Erdembileg Anuurad,
Wei Zhang,
Chin-Shang Li,
Robert C. Kaplan,
Jason Lazar,
Dan Merenstein,
Roksana Karim,
Brad Aouizerat,
Mardge H. Cohen,
Kenneth R. Butler,
Savita Pahwa,
Igho Ofotokun,
Adaora A. Adimora,
Elizabeth T. Golub,
Lars Berglund
Publication year - 2017
Publication title -
arteriosclerosis thrombosis and vascular biology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 3.007
H-Index - 270
eISSN - 1524-4636
pISSN - 1079-5642
DOI - 10.1161/atvbaha.117.309137
Subject(s) - human immunodeficiency virus (hiv) , lipoprotein(a) , medicine , lipoprotein , biology , cholesterol , immunology
In the general population, lipoprotein(a) [Lp(a)] has been established as an independent causal risk factor for cardiovascular disease. Lp(a) levels are to a major extent regulated by a size polymorphism in the apolipoprotein(a) [apo(a)] gene. The roles of Lp(a)/apo(a) in human immunodeficiency virus (HIV)-related elevated cardiovascular disease risk remain unclear.
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