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Nucleotide P2Y 2 receptor (P2Y 2 R) contributes to vascular inflammation by increasing vascular cell adhesion molecule-1 expression in endothelial cells (EC), and global P2Y 2 R deficiency prevents fatty streak formation in apolipoprotein E null (ApoE -/- ) mice. Because P2Y 2 R is ubiquitously expressed in vascular cells, we investigated the contribution of endothelial P2Y 2 R in the pathogenesis of atherosclerosis.

Endothelial Cell–Specific Deletion of P2Y 2 Receptor Promotes Plaque Stability in Atherosclerosis-Susceptible ApoE-Null Mice

Endothelial Cell–Specific Deletion of P2Y ₂ Receptor Promotes Plaque Stability in Atherosclerosis-Susceptible ApoE-Null Mice