RasGRF Couples Nox4-Dependent Endoplasmic Reticulum Signaling to Ras | Zendy

Ru Feng Wu | Zendy; Chengxu Liao | Zendy; Hadi Hatoum | Zendy; Guosheng Fu | Zendy; Cristhiaan D. Ochoa | Zendy; Lance S. Terada | Zendy

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RasGRF Couples Nox4-Dependent Endoplasmic Reticulum Signaling to Ras

Author(s) -

Ru Feng Wu,

Chengxu Liao,

Hadi Hatoum,

Guosheng Fu,

Cristhiaan D. Ochoa,

Lance S. Terada

Publication year - 2016

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.116.307922

Subject(s) - unfolded protein response , endoplasmic reticulum , microbiology and biotechnology , nox4 , tunicamycin , calcium pump , nicotinamide adenine dinucleotide phosphate , signal transduction , calcium signaling , nadph oxidase , chemistry , calcium , reactive oxygen species , biology , biochemistry , oxidase test , atpase , enzyme , organic chemistry

In response to endoplasmic reticulum (ER) stress, endothelial cells initiate corrective pathways such as the unfolded protein response. Recent studies suggest that reactive oxygen species produced on the ER may participate in homeostatic signaling through Ras in response to ER stress. We sought to identify mechanisms responsible for this focal signaling pathway.

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