Deletion of Hyaluronan Synthase 3 Inhibits Neointimal Hyperplasia in Mice | Zendy

Lena S. Kiene | Zendy; Susanne Homann | Zendy; Tatsiana Suvorava | Zendy; Berit Rabausch | Zendy; Julia Müller | Zendy; Georg Kojda | Zendy; Inga Kretschmer | Zendy; Sören Twarock | Zendy; Guang Dai | Zendy; René Deenen | Zendy; Sonja Hartwig | Zendy; Stefan Lehr | Zendy; Karl Köhrer | Zendy; Rashmin C. Savani | Zendy; Maria Grandoch | Zendy; Jens W. Fischer | Zendy

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Deletion of Hyaluronan Synthase 3 Inhibits Neointimal Hyperplasia in Mice

Author(s) -

Lena S. Kiene,

Susanne Homann,

Tatsiana Suvorava,

Berit Rabausch,

Julia Müller,

Georg Kojda,

Inga Kretschmer,

Sören Twarock,

Guang Dai,

René Deenen,

Sonja Hartwig,

Stefan Lehr,

Karl Köhrer,

Rashmin C. Savani,

Maria Grandoch,

Jens W. Fischer

Publication year - 2016

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.115.306607

Subject(s) - hyaluronan synthase , neointimal hyperplasia , pericyte , neointima , extracellular matrix , chemistry , microbiology and biotechnology , vascular smooth muscle , endothelial stem cell , biology , endocrinology , medicine , biochemistry , in vitro , restenosis , stent , smooth muscle

Hyaluronan (HA) is a polymeric glucosaminoglycan that forms a provisional extracellular matrix in diseased vessels. HA is synthesized by 3 different HA synthases (HAS1, HAS2, and HAS3). Aim of this study was to unravel the role of the HAS3 isoenzyme during experimental neointimal hyperplasia.

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