S100A6 Regulates Endothelial Cell Cycle Progression by Attenuating Antiproliferative Signal Transducers and Activators of Transcription 1 Signaling
Author(s) -
Carolin Lerchenmüller,
Julian Heißenberg,
Federico Damilano,
Vassilios J. Bezzeridis,
Isabel Krämer,
MarieLuce BochatonPiallat,
Kristóf Hirschberg,
Martin Busch,
Hugo A. Katus,
Karsten Peppel,
Anthony Rosenzweig,
Hauke Busch,
Melanie Boerries,
Patrick Most
Publication year - 2016
Publication title -
arteriosclerosis thrombosis and vascular biology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 3.007
H-Index - 270
eISSN - 1524-4636
pISSN - 1079-5642
DOI - 10.1161/atvbaha.115.306415
Subject(s) - signal transduction , microbiology and biotechnology , cell cycle progression , transcription (linguistics) , cell cycle , transcription factor , endothelial stem cell , cancer research , biology , cell , medicine , chemistry , genetics , in vitro , gene , linguistics , philosophy
S100A6, a member of the S100 protein family, has been described as relevant for cell cycle entry and progression in endothelial cells. The molecular mechanism conferring S100A6's proliferative actions, however, remained elusive.
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