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Angiotensin II (Ang II) infusion causes aortic medial thickening via stimulation of angiotensin II type 1a (AT1a) receptors. The purpose of this study was to determine the cellular loci of AT1a receptors that mediate this Ang II-induced aortic pathology.

Fibroblast Angiotensin II Type 1a Receptors Contribute to Angiotensin II–Induced Medial Hyperplasia in the Ascending Aorta