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In resistance arteries, there is an emerging view that smooth muscle CaV3.2 channels restrain arterial constriction through a feedback response involving the large-conductance Ca(2+)-activated K(+) channel (BKCa). Here, we used wild-type and CaV3.2 knockout (CaV3.2(-/-)) mice to definitively test whether CaV3.2 moderates myogenic tone in mesenteric arteries via the CaV3.2-ryanodine receptor-BKCa axis and whether this regulatory mechanism influences blood pressure regulation.

Genetic Ablation of Ca V 3.2 Channels Enhances the Arterial Myogenic Response by Modulating the RyR-BK Ca Axis

Genetic Ablation of Ca _V 3.2 Channels Enhances the Arterial Myogenic Response by Modulating the RyR-BK _Ca Axis