Mediation of Cardiovascular Risk Factor Effects Through Subclinical Vascular Disease | Zendy

Joseph Yeboah | Zendy; Joseph A. Delaney | Zendy; Robin M. Nance | Zendy; Robyn L. McClelland | Zendy; Joseph F. Polak | Zendy; Christopher T. Sibley | Zendy; Alain G. Bertoni | Zendy; Gregory L. Burke | Zendy; J. Jeffrey Carr | Zendy; David M. Herrington | Zendy

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Mediation of Cardiovascular Risk Factor Effects Through Subclinical Vascular Disease

Author(s) -

Joseph Yeboah,

Joseph A. Delaney,

Robin M. Nance,

Robyn L. McClelland,

Joseph F. Polak,

Christopher T. Sibley,

Alain G. Bertoni,

Gregory L. Burke,

J. Jeffrey Carr,

David M. Herrington

Publication year - 2014

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.114.303753

Subject(s) - medicine , body mass index , mediation , disease , confidence interval , subclinical infection , cardiology , brachial artery , risk factor , intima media thickness , demography , carotid arteries , blood pressure , sociology , political science , law

It is unclear to what extent subclinical cardiovascular disease (CVD) such as coronary artery calcium (CAC), carotid intima-media thickness (CIMT), and brachial flow-mediated dilation (FMD) are mediators of the known associations between traditional cardiovascular risk factors and incident CVD events. We assessed the portion of the effects of risk factors on incident CVD events that are mediated through CAC, CIMT, and FMD.

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