S100/Calgranulin-Mediated Inflammation Accelerates Left Ventricular Hypertrophy and Aortic Valve Sclerosis in Chronic Kidney Disease in a Receptor for Advanced Glycation End Products–Dependent Manner | Zendy

Ling Yan | Zendy; Liby Mathew | Zendy; Bijoy Chellan | Zendy; Brandon Gardner | Zendy; Judy U. Earley | Zendy; Tipu S. Puri | Zendy; Marion A. Hofmann Bowman | Zendy

Open Access

S100/Calgranulin-Mediated Inflammation Accelerates Left Ventricular Hypertrophy and Aortic Valve Sclerosis in Chronic Kidney Disease in a Receptor for Advanced Glycation End Products–Dependent Manner

Author(s) -

Ling Yan,

Liby Mathew,

Bijoy Chellan,

Brandon Gardner,

Judy U. Earley,

Tipu S. Puri,

Marion A. Hofmann Bowman

Publication year - 2014

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.114.303508

Subject(s) - medicine , kidney disease , fibroblast growth factor 23 , endocrinology , parathyroid hormone , calcium

S100A12 and fibroblast growth factor 23 are biomarkers of cardiovascular morbidity and mortality in patients with chronic kidney disease (CKD). We tested the hypothesis that human S100/calgranulin would accelerate cardiovascular disease in mice subjected to CKD.

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