Loss of Id3 Increases VCAM-1 Expression, Macrophage Accumulation, and Atherogenesis in Ldlr –/– Mice | Zendy

Michael J. Lipinski | Zendy; Kirsti A. Campbell | Zendy; Son Q. Duong | Zendy; Thomas Welch | Zendy; James C. Garmey | Zendy; Amanda C. Doran | Zendy; Marcus D. Skaflen | Zendy; Stephanie Oldham | Zendy; Kimberly M. Kelly | Zendy; Coleen A. McNamara | Zendy

AI Assistant Blog Pricing

Home ZAIA Blog

Open Access

Loss of Id3 Increases VCAM-1 Expression, Macrophage Accumulation, and Atherogenesis in Ldlr ^–/– Mice

Author(s) -

Michael J. Lipinski,

Kirsti A. Campbell,

Son Q. Duong,

Thomas Welch,

James C. Garmey,

Amanda C. Doran,

Marcus D. Skaflen,

Stephanie Oldham,

Kimberly M. Kelly,

Coleen A. McNamara

Publication year - 2012

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.112.300352

Subject(s) - ldl receptor , vcam 1 , biology , immunology , microbiology and biotechnology , endocrinology , cell adhesion molecule , lipoprotein , cholesterol , icam 1

Inhibitor of differention-3 (Id3) promotes B cells homing to the aorta and atheroprotection in Apoe(-/-) mice. We sought to determine the impact of loss of Id3 in the Ldlr((-/-)) mouse model of diet-induced atherosclerosis and identify novel Id3 targets in the vessel wall.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research