MyD88 Deficiency Attenuates Angiotensin II-Induced Abdominal Aortic Aneurysm Formation Independent of Signaling Through Toll-Like Receptors 2 and 4 | Zendy

A. Phillip Owens | Zendy; Debra L. Rateri | Zendy; Deborah A. Howatt | Zendy; Kathryn J. Moore | Zendy; Peter S. Tobias | Zendy; Linda K. Curtiss | Zendy; Hong Lü | Zendy; Lisa A. Cassis | Zendy; Alan Daugherty | Zendy

Open Access

MyD88 Deficiency Attenuates Angiotensin II-Induced Abdominal Aortic Aneurysm Formation Independent of Signaling Through Toll-Like Receptors 2 and 4

Author(s) -

A. Phillip Owens,

Debra L. Rateri,

Deborah A. Howatt,

Kathryn J. Moore,

Peter S. Tobias,

Linda K. Curtiss,

Hong Lü,

Lisa A. Cassis,

Alan Daugherty

Publication year - 2011

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.111.238642

Subject(s) - angiotensin ii , tlr2 , tlr4 , bone marrow , medicine , endocrinology , receptor , ldl receptor , immunology , lipoprotein , cholesterol

The purpose of this study was to determine whether myeloid differentiation factor 88 (MyD88) and its related Toll-like receptors (TLRs) 2 and 4 contributed to the development of angiotensin II (AngII)-induced abdominal aortic aneurysms (AAAs) and atherosclerosis.

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