Nucleotide-Binding Oligomerization Domain Protein 2 Deficiency Enhances Neointimal Formation in Response to Vascular Injury | Zendy

Min-Young Kwon | Zendy; Xiaoli Liu | Zendy; SeonJin Lee | Zendy; Young-Ho Kang | Zendy; Augustine M.K. Choi | Zendy; KiUp Lee | Zendy; Mark A. Perrella | Zendy; Su Wol Chung | Zendy

Open Access

Nucleotide-Binding Oligomerization Domain Protein 2 Deficiency Enhances Neointimal Formation in Response to Vascular Injury

Author(s) -

Min-Young Kwon,

Xiaoli Liu,

SeonJin Lee,

Young-Ho Kang,

Augustine M.K. Choi,

KiUp Lee,

Mark A. Perrella,

Su Wol Chung

Publication year - 2011

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.111.235135

Subject(s) - vascular smooth muscle , nod2 , neointimal hyperplasia , muramyl dipeptide , neointima , cancer research , microbiology and biotechnology , chemistry , biology , endocrinology , medicine , biochemistry , restenosis , receptor , in vitro , smooth muscle , innate immune system , stent

Nucleotide-binding oligomerization domain protein 2 (NOD2) stimulates diverse inflammatory responses resulting in differential cellular phenotypes. To identify the role of NOD2 in vascular arterial obstructive diseases, we investigated the expression and pathophysiological role of NOD2 in a vascular injury model of neointimal hyperplasia.

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