Left Atrial Blood Stasis and Von Willebrand Factor–ADAMTS13 Homeostasis in Atrial Fibrillation | Zendy

Naser M. Ammash | Zendy; Ewa Konik | Zendy; Robert D. McBane | Zendy; Dong Chen | Zendy; Julie I Tange | Zendy; Diane E. Grill | Zendy; Regina M. Herges | Zendy; Thomas G. McLeod | Zendy; Paul A. Friedman | Zendy; Waldemar E. Wysokiński | Zendy

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Left Atrial Blood Stasis and Von Willebrand Factor–ADAMTS13 Homeostasis in Atrial Fibrillation

Author(s) -

Naser M. Ammash,

Ewa Konik,

Robert D. McBane,

Dong Chen,

Julie I Tange,

Diane E. Grill,

Regina M. Herges,

Thomas G. McLeod,

Paul A. Friedman,

Waldemar E. Wysokiński

Publication year - 2011

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.111.232991

Subject(s) - von willebrand factor , atrial fibrillation , medicine , cardiology , blood stasis , sinus rhythm , adamts13 , thrombus , platelet , pathology , alternative medicine , traditional chinese medicine

Left atrial blood stasis is associated with increased risk for left atrial appendage thrombus (LAAT) and stroke in atrial fibrillation (AF). Von Willebrand factor (VWF) is associated with thromboembolism in AF. VWF thrombogenic activity is proportional to multimer size, which is regulated by VWF-cleaving protease (ADAMTS13).

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