Induction of Smooth Muscle Cell Migration During Arteriogenesis Is Mediated by Rap2
Author(s) -
Jochen Pöling,
Marten Szibor,
Silvia Schimanski,
Marie-Elisabeth Ingelmann,
Wolfgang Rees,
Praveen Gajawada,
Zaber Kochfar,
Holger Lörchner,
Isabelle Salwig,
Jaeyoung Shin,
Karsten Wiebe,
Thomas Kubin,
H. Warnecke,
Thomas Braun
Publication year - 2011
Publication title -
arteriosclerosis thrombosis and vascular biology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 3.007
H-Index - 270
eISSN - 1524-4636
pISSN - 1079-5642
DOI - 10.1161/atvbaha.111.232835
Subject(s) - arteriogenesis , downregulation and upregulation , vascular smooth muscle , microbiology and biotechnology , fibroblast growth factor receptor 1 , fibroblast growth factor , receptor , chemistry , cancer research , medicine , biology , endocrinology , angiogenesis , smooth muscle , biochemistry , gene
Collateral artery growth or arteriogenesis is the primary means of the circulatory system to maintain blood flow in the face of major arterial occlusions. Arteriogenesis depends on activation of fibroblast growth factor (FGF) receptors, but relatively little is known about downstream mediators of FGF signaling.
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