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Response Gene to Complement 32 Promotes Vascular Lesion Formation Through Stimulation of Smooth Muscle Cell Proliferation and Migration
Author(s) -
JiaNing Wang,
Ning Shi,
WeiBing Xie,
Xia Guo,
ShiYou Chen
Publication year - 2011
Publication title -
arteriosclerosis thrombosis and vascular biology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 3.007
H-Index - 270
eISSN - 1524-4636
pISSN - 1079-5642
DOI - 10.1161/atvbaha.111.230706
Subject(s) - neointima , vascular smooth muscle , microbiology and biotechnology , biology , gene knockdown , cell growth , downregulation and upregulation , cell migration , pathology , cell , endocrinology , medicine , restenosis , apoptosis , gene , biochemistry , smooth muscle , stent
The objectives of this study were to determine the role of response gene to complement 32 (RGC-32) in vascular lesion formation after experimental angioplasty and to explore the underlying mechanisms.

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