Targeted Disruption of the Prostaglandin E 2 E-Prostanoid 2 Receptor Exacerbates Vascular Neointimal Formation in Mice | Zendy

Sen Zhu | Zendy; Rui Xue | Zendy; Pan Zhao | Zendy; Fenling Fan | Zendy; Xiaomu Kong | Zendy; Senfeng Zheng | Zendy; Qifei Han | Zendy; Yi Zhu | Zendy; Nanping Wang | Zendy; Jichun Yang | Zendy; Youfei Guan | Zendy

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Targeted Disruption of the Prostaglandin E ₂ E-Prostanoid 2 Receptor Exacerbates Vascular Neointimal Formation in Mice

Author(s) -

Sen Zhu,

Rui Xue,

Pan Zhao,

Fenling Fan,

Xiaomu Kong,

Senfeng Zheng,

Qifei Han,

Yi Zhu,

Nanping Wang,

Jichun Yang,

Youfei Guan

Publication year - 2011

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.111.226142

Subject(s) - prostanoid , receptor , endocrinology , medicine , prostaglandin , chemistry , microbiology and biotechnology , biology

Restenosis after angioplasty remains a major clinical problem. Prostaglandin E(2) (PGE(2)) plays an important role in vascular homeostasis. The PGE(2) receptor E-prostanoid 2 (EP2) is involved in the proliferation and migration of various cell types. We aimed to determine the role of EP2 in the pathogenesis of neointimal formation after vascular injury.

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