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Despite the fact that mechanical stresses are well recognized as key determinants for atherosclerotic plaque rupture, very little is known about stress amplitude and distribution in atherosclerotic lesions, even in the standard apolipoprotein E (apoE)-/- mouse model of atherosclerosis. Our objectives were to combine immunohistology, atomic force microscopy measurements, and finite element computational analysis for the accurate quantification of stress amplitude and distribution in apoE-/- mouse aortic atherosclerotic lesions.

Assessing Low Levels of Mechanical Stress in Aortic Atherosclerotic Lesions From Apolipoprotein E −/− Mice—Brief Report

Assessing Low Levels of Mechanical Stress in Aortic Atherosclerotic Lesions From Apolipoprotein E ^−/− Mice—Brief Report