Cilostazol Suppresses Angiotensin II–Induced Vasoconstriction via Protein Kinase A–Mediated Phosphorylation of the Transient Receptor Potential Canonical 6 Channel | Zendy

Kinue Nishioka | Zendy; Motohiro Nishida | Zendy; Marina Ariyoshi | Zendy; Zhong Jian | Zendy; Shota Saiki | Zendy; Mayumi Hirano | Zendy; Michio Nakaya | Zendy; Yoji Sato | Zendy; Satomi Kita | Zendy; Takahiro Iwamoto | Zendy; Katsuya Hirano | Zendy; Ryuji Inoue | Zendy; Hitoshi Kurose | Zendy

Open Access

Cilostazol Suppresses Angiotensin II–Induced Vasoconstriction via Protein Kinase A–Mediated Phosphorylation of the Transient Receptor Potential Canonical 6 Channel

Author(s) -

Kinue Nishioka,

Motohiro Nishida,

Marina Ariyoshi,

Zhong Jian,

Shota Saiki,

Mayumi Hirano,

Michio Nakaya,

Yoji Sato,

Satomi Kita,

Takahiro Iwamoto,

Katsuya Hirano,

Ryuji Inoue,

Hitoshi Kurose

Publication year - 2011

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.110.221010

Subject(s) - transient receptor potential channel , vasoconstriction , chemistry , phosphorylation , cilostazol , transient (computer programming) , protein kinase a , angiotensin ii , trpc1 , receptor , pharmacology , microbiology and biotechnology , endocrinology , medicine , biology , biochemistry , computer science , aspirin , operating system

The goal of this study was to determine whether inhibition of transient receptor potential canonical (TRPC) channels underlies attenuation of angiotensin II (Ang II)-induced vasoconstriction by phosphodiesterase (PDE) 3 inhibition.

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