RhoJ/TCL Regulates Endothelial Motility and Tube Formation and Modulates Actomyosin Contractility and Focal Adhesion Numbers | Zendy

Sukhbir Kaur | Zendy; Katarzyna B. Leszczyńska | Zendy; Sabu Abraham | Zendy; Margherita Scarcia | Zendy; Sabina Hiltbrunner | Zendy; Christopher J. Marshall | Zendy; Georgia Mavria | Zendy; Roy Bicknell | Zendy; Victoria L. Heath | Zendy

Open Access

RhoJ/TCL Regulates Endothelial Motility and Tube Formation and Modulates Actomyosin Contractility and Focal Adhesion Numbers

Author(s) -

Sukhbir Kaur,

Katarzyna B. Leszczyńska,

Sabu Abraham,

Margherita Scarcia,

Sabina Hiltbrunner,

Christopher J. Marshall,

Georgia Mavria,

Roy Bicknell,

Victoria L. Heath

Publication year - 2010

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.110.216341

Subject(s) - focal adhesion , microbiology and biotechnology , motility , contractility , endothelial stem cell , biology , myosin , vascular endothelial growth factor b , myosin light chain kinase , chemistry , vascular endothelial growth factor a , vascular endothelial growth factor , signal transduction , endocrinology , cancer research , biochemistry , in vitro , vegf receptors

RhoJ/TCL was identified by our group as an endothelial-expressed Rho GTPase. The aim of this study was to determine its tissue distribution, subcellular localization, and function in endothelial migration and tube formation.

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