Protease-Activated Receptor 2 Deficiency Reduces Cardiac Ischemia/Reperfusion Injury | Zendy

Silvio Antoniak | Zendy; Mauricio Rojas | Zendy; Denise J. Spring | Zendy; Tara A. Bullard | Zendy; Edward D. Verrier | Zendy; Burns C. Blaxall | Zendy; Nigel Mackman | Zendy; Rafał Pawliński | Zendy

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Protease-Activated Receptor 2 Deficiency Reduces Cardiac Ischemia/Reperfusion Injury

Author(s) -

Silvio Antoniak,

Mauricio Rojas,

Denise J. Spring,

Tara A. Bullard,

Edward D. Verrier,

Burns C. Blaxall,

Nigel Mackman,

Rafał Pawliński

Publication year - 2010

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.110.213280

Subject(s) - reperfusion injury , medicine , ischemia , myocardial infarction , inflammation , proinflammatory cytokine , cardiac function curve , oxidative stress , endocrinology , ventricular remodeling , phosphorylation , receptor , cardiology , biology , heart failure , biochemistry

To investigate the effect of protease-activated receptor (PAR) 2 deficiency on ischemia/reperfusion (I/R) injury-induced infarct size, inflammation, heart remodeling, and cardiac function.

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