C-Reactive Protein-Mediated Vascular Injury Requires Complement | Zendy

Fadi G. Hage | Zendy; Suzanne Oparil | Zendy; Dongqi Xing | Zendy; Yiu-Fai Chen | Zendy; Mark A. McCrory | Zendy; Alexander J. Szalai | Zendy

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C-Reactive Protein-Mediated Vascular Injury Requires Complement

Author(s) -

Fadi G. Hage,

Suzanne Oparil,

Dongqi Xing,

Yiu-Fai Chen,

Mark A. McCrory,

Alexander J. Szalai

Publication year - 2010

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.110.205377

Subject(s) - neointima , adventitia , c reactive protein , genetically modified mouse , medicine , ligation , complement system , blood vessel , pathology , common carotid artery , inflammation , immunology , biology , transgene , carotid arteries , restenosis , antibody , biochemistry , stent , gene

We previously demonstrated that vascular injury-induced neointima formation is exaggerated in human C-reactive protein (CRP) transgenic (CRPtg) compared to nontransgenic (NTG) mice. We now test the hypothesis that complement is required for this effect.

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