Monocyte Chemoattractant Proteins Mediate Myocardial Microvascular Dysfunction in Swine Renovascular Hypertension | Zendy

Jing Lin | Zendy; Xiangyang Zhu | Zendy; Alejandro Chade | Zendy; Kyra L. Jordan | Zendy; Ronit Lavi | Zendy; Elena Daghini | Zendy; Matthew E. Gibson | Zendy; Angelo Guglielmotti | Zendy; Amir Lerman | Zendy; Lilach O. Lerman | Zendy

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Monocyte Chemoattractant Proteins Mediate Myocardial Microvascular Dysfunction in Swine Renovascular Hypertension

Author(s) -

Jing Lin,

Xiangyang Zhu,

Alejandro Chade,

Kyra L. Jordan,

Ronit Lavi,

Elena Daghini,

Matthew E. Gibson,

Angelo Guglielmotti,

Amir Lerman,

Lilach O. Lerman

Publication year - 2009

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.109.190546

Subject(s) - medicine , cardiology , fibrosis , inflammation , renovascular hypertension , myocardial fibrosis , endocrinology , blood pressure

Monocyte chemoattractant proteins (MCPs) play an important role in mediating inflammatory processes. Hypertension (HTN) is associated with inflammation as well as impaired cardiac microcirculatory function and structure, but the contribution of MCPs to these alterations remained unclear. This study tested the hypothesis that MCPs regulate cardiac microvascular function and structure in experimental HTN.

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