Role of the Low-Affinity Leukotriene B 4 Receptor BLT2 in VEGF-Induced Angiogenesis | Zendy

Geun-Young Kim | Zendy; Jinwook Lee | Zendy; Sung-Hoon Cho | Zendy; Ji Min Seo | Zendy; JaeHong Kim | Zendy

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Role of the Low-Affinity Leukotriene B ₄ Receptor BLT2 in VEGF-Induced Angiogenesis

Author(s) -

Geun-Young Kim,

Jinwook Lee,

Sung-Hoon Cho,

Ji Min Seo,

JaeHong Kim

Publication year - 2009

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.109.185793

Subject(s) - angiogenesis , matrigel , chemistry , vascular endothelial growth factor , gene knockdown , umbilical vein , in vivo , microbiology and biotechnology , pharmacology , cancer research , biology , in vitro , biochemistry , vegf receptors , apoptosis

The leukotriene B(4) (LTB(4)) receptor BLT2 is expressed in endothelium, but no clear physiological function for it has yet been identified, especially in vascular angiogenesis. The purpose of this study is to characterize the potential function of BLT2 in vascular endothelial growth factor (VEGF)-induced angiogenesis.

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