Endothelial Nitric Oxide Synthase Inhibits G 12/13 and Rho-Kinase Activated by the Angiotensin II Type-1 Receptor | Zendy

Hiroyuki Suzuki | Zendy; Keita Kimura | Zendy; Heigoro Shirai | Zendy; Kunie Eguchi | Zendy; Sadaharu Higuchi | Zendy; Akinari Hinoki | Zendy; Kazuhiro Ishimaru | Zendy; Eugen Brailoiu | Zendy; Danny N. Dhanasekaran | Zendy; Laura N. Stemmle | Zendy; Timothy A. Fields | Zendy; Gerald D. Frank | Zendy; Michael V. Autieri | Zendy; Satoru Eguchi | Zendy

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Endothelial Nitric Oxide Synthase Inhibits G _12/13 and Rho-Kinase Activated by the Angiotensin II Type-1 Receptor

Author(s) -

Hiroyuki Suzuki,

Keita Kimura,

Heigoro Shirai,

Kunie Eguchi,

Sadaharu Higuchi,

Akinari Hinoki,

Kazuhiro Ishimaru,

Eugen Brailoiu,

Danny N. Dhanasekaran,

Laura N. Stemmle,

Timothy A. Fields,

Gerald D. Frank,

Michael V. Autieri,

Satoru Eguchi

Publication year - 2008

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.108.181024

Subject(s) - enos , angiotensin ii , transactivation , vascular smooth muscle , signal transduction , nitric oxide synthase , nitric oxide synthase type iii , microbiology and biotechnology , receptor , chemistry , medicine , nitric oxide , endocrinology , biology , biochemistry , gene expression , gene , smooth muscle

Although, endothelial nitric oxide (NO) synthase (eNOS) is believed to antagonize vascular remodeling induced by the angiotensin II (AngII) type-1 receptor, the exact signaling mechanism remains unclear.

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