Coexpression of CLA-1 and Human PDZK1 in Murine Liver Modulates HDL Cholesterol Metabolism | Zendy

Hidenori Komori | Zendy; Hidenori Arai | Zendy; Terumi Kashima | Zendy; Thierry Huby | Zendy; Toru Kita | Zendy; Yukihiko Ueda | Zendy

AI Assistant Blog Pricing

Home ZAIA Blog

Open Access

Coexpression of CLA-1 and Human PDZK1 in Murine Liver Modulates HDL Cholesterol Metabolism

Author(s) -

Hidenori Komori,

Hidenori Arai,

Terumi Kashima,

Thierry Huby,

Toru Kita,

Yukihiko Ueda

Publication year - 2008

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.108.165845

Subject(s) - reverse cholesterol transport , cd36 , cholesterol , scavenger receptor , genetically modified mouse , transgene , cholesterylester transfer protein , abca1 , biology , endocrinology , liver x receptor , cholesteryl ester , medicine , high density lipoprotein , lipoprotein , in vivo , metabolism , chemistry , receptor , biochemistry , gene , nuclear receptor , transporter , transcription factor , microbiology and biotechnology

In rodents scavenger receptor class B type I (SR-BI) is a key molecule for selective uptake of cholesteryl ester from high-density lipoprotein (HDL). This study was aimed to clarify the role of the human SR-BI/CD36 and LIMP-II Analogues-1 (CLA-1) as a molecular target of selective uptake of cholesteryl ester from HDL in vivo.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research