Toll-Like Receptors 2-Deficient Mice Are Protected Against Postischemic Coronary Endothelial Dysfunction | Zendy

Julie Favre | Zendy; Philippe Musette | Zendy; Victorine DouinEchinard | Zendy; Karine Laude | Zendy; JeanPaul Henry | Zendy; JeanFrançois Arnal | Zendy; Christian Thuillez | Zendy; Vincent Richard | Zendy

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Toll-Like Receptors 2-Deficient Mice Are Protected Against Postischemic Coronary Endothelial Dysfunction

Author(s) -

Julie Favre,

Philippe Musette,

Victorine DouinEchinard,

Karine Laude,

JeanPaul Henry,

JeanFrançois Arnal,

Christian Thuillez,

Vincent Richard

Publication year - 2007

Publication title -

arteriosclerosis thrombosis and vascular biology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.007

H-Index - 270

eISSN - 1524-4636

pISSN - 1079-5642

DOI - 10.1161/atvbaha.107.140723

Subject(s) - tlr2 , bone marrow , endothelial dysfunction , ischemia , endothelial stem cell , medicine , receptor , reperfusion injury , immunology , biology , chemistry , endocrinology , innate immune system , in vitro , biochemistry

Toll-like receptors (TLR) 2 are expressed in cardiac and inflammatory cells, and regulate leukocyte function. Because leukocyte adhesion is a critical event in endothelial injury induced by ischemia/reperfusion (I/R), we assessed whether TLR2 were involved in I/R-induced coronary endothelial injury.

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