Neurological Deterioration in Acute Lacunar Infarctions | Zendy

Joaquı́n Serena | Zendy; Rogelio Leira | Zendy; José Castillo | Zendy; José Manuel Pumar | Zendy; Mar Castellanos | Zendy; Antoni Dávalos | Zendy

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Neurological Deterioration in Acute Lacunar Infarctions

Author(s) -

Joaquı́n Serena,

Rogelio Leira,

José Castillo,

José Manuel Pumar,

Mar Castellanos,

Antoni Dávalos

Publication year - 2001

Publication title -

stroke

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.397

H-Index - 319

eISSN - 1524-4628

pISSN - 0039-2499

DOI - 10.1161/01.str.32.5.1154

Subject(s) - glutamate receptor , medicine , lacunar stroke , excitotoxicity , stroke (engine) , cardiology , excitatory amino acid transporter , central nervous system disease , magnetic resonance imaging , pathophysiology , biomarker , gastroenterology , anesthesia , ischemic stroke , ischemia , radiology , biochemistry , mechanical engineering , receptor , engineering , chemistry

The mechanisms involved in the neurological deterioration of acute lacunar strokes are unknown. Although accumulating evidence suggests that glutamate release plays a role in the progression of territorial infarctions, it remains to be established whether excitotoxicity also participates in lacunar stroke progression. We investigated whether excitatory and inhibitory amino acid concentrations in blood predict subsequent progressive motor deficits in lacunar infarctions.

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