Methamphetamine Potentiates Ischemia/Reperfusion Insults After Transient Middle Cerebral Artery Ligation | Zendy

Yun Wang | Zendy; Teruo Hayashi | Zendy; ChenFu Chang | Zendy; YungHsiao Chiang | Zendy; Li-I Tsao | Zendy; TsungPing Su | Zendy; Cesar V. Borlongan | Zendy; ShinnZong Lin | Zendy

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Methamphetamine Potentiates Ischemia/Reperfusion Insults After Transient Middle Cerebral Artery Ligation

Author(s) -

Yun Wang,

Teruo Hayashi,

ChenFu Chang,

YungHsiao Chiang,

Li-I Tsao,

TsungPing Su,

Cesar V. Borlongan,

ShinnZong Lin

Publication year - 2001

Publication title -

stroke

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.397

H-Index - 319

eISSN - 1524-4628

pISSN - 0039-2499

DOI - 10.1161/01.str.32.3.775

Subject(s) - glial cell line derived neurotrophic factor , medicine , ischemia , pharmacology , neuroprotection , anesthesia , neurotrophic factors , infarction , striatum , middle cerebral artery , stroke (engine) , dopamine , receptor , mechanical engineering , engineering , myocardial infarction

Previous studies have indicated that both methamphetamine (MA) and ischemia/reperfusion injuries involve reactive oxygen species formation and activation of apoptotic mechanism. That MA could have a synergistic or additive effect with stroke-induced brain damage is possible. The purpose of the present study was to investigate whether administration of MA in vivo would potentiate ischemic brain injury.

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