Intracisternal Increase of Superoxide Anion Production in a Canine Subarachnoid Hemorrhage Model | Zendy

Takashi Mori | Zendy; Kazuya Nagata | Zendy; Terrence Town | Zendy; Jun Tan | Zendy; Toru Matsui | Zendy; Takao Asano | Zendy

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Intracisternal Increase of Superoxide Anion Production in a Canine Subarachnoid Hemorrhage Model

Author(s) -

Takashi Mori,

Kazuya Nagata,

Terrence Town,

Jun Tan,

Toru Matsui,

Takao Asano

Publication year - 2001

Publication title -

stroke

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.397

H-Index - 319

eISSN - 1524-4628

pISSN - 0039-2499

DOI - 10.1161/01.str.32.3.636

Subject(s) - subarachnoid hemorrhage , superoxide , medicine , subarachnoid space , basilar artery , cerebral vasospasm , pathology , superoxide dismutase , reactive oxygen species , nad(p)h oxidase , vasospasm , adventitia , colocalization , nadph oxidase , microbiology and biotechnology , oxidative stress , biochemistry , chemistry , biology , cerebrospinal fluid , enzyme

Reactive oxygen species (ROS) are thought to be primary in the pathogenesis of cerebral vasospasm after subarachnoid hemorrhage (SAH). However, as direct evidence of ROS has not yet been demonstrated in cerebral vasospasm, we sought to substantiate superoxide anion (.O(2)(-)) generation in the subarachnoid space after SAH using a modification of Karnovsky's manganese/diaminobenzidine (Mn(2+)/DAB) technique.

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