Reduced Cerebrovascular CO 2 Reactivity in CADASIL | Zendy

Thomas Pfefferkorn | Zendy; Sebastian von Stuckrad-Barre | Zendy; Jürgen Herzog | Zendy; Thomas Gasser | Zendy; Gerhard F. Hamann | Zendy; Martin Dichgans | Zendy

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Reduced Cerebrovascular CO ₂ Reactivity in CADASIL

Author(s) -

Thomas Pfefferkorn,

Sebastian von Stuckrad-Barre,

Jürgen Herzog,

Thomas Gasser,

Gerhard F. Hamann,

Martin Dichgans

Publication year - 2001

Publication title -

stroke

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.397

H-Index - 319

eISSN - 1524-4628

pISSN - 0039-2499

DOI - 10.1161/01.str.32.1.17

Subject(s) - cadasil , medicine , angiopathy , transcranial doppler , cerebral arteries , cardiology , middle cerebral artery , leukoencephalopathy , hemodynamics , endocrinology , ischemia , disease , diabetes mellitus

Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukencephalopathy (CADASIL) is a hereditary angiopathy caused by mutations in Notch3. Cerebral microvessels show an accumulation of granular osmiophilic material in the vicinity of degenerating vascular smooth muscle cells. To study cerebrovascular function in CADASIL, we performed measurements on cerebral hemodynamics by using transcranial Doppler sonography.

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