z-logo
open-access-imgOpen Access
Are Morphological or Functional Changes in the Carotid Artery Wall Associated With Chlamydia pneumoniae, Helicobacter pylori, Cytomegalovirus, or Herpes Simplex Virus Infection?
Author(s) -
Christine EspinolaKlein,
HansJürgen Rupprecht,
Stefan Blankenberg,
Christoph Bickel,
Helmuth Kopp,
Gerd Rippin,
Gerd Häfner,
Ulf Pfeifer,
Jürgen Meyer
Publication year - 2000
Publication title -
stroke
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 3.397
H-Index - 319
eISSN - 1524-4628
pISSN - 0039-2499
DOI - 10.1161/01.str.31.9.2127
Subject(s) - medicine , chlamydia , helicobacter pylori , herpes simplex virus , intima media thickness , stenosis , cytomegalovirus , pathogenesis , gastroenterology , chlamydophila pneumoniae , cardiology , serology , immunology , antibody , virus , carotid arteries , herpesviridae , chlamydiaceae , viral disease
Chronic infection with Chlamydia pneumoniae, Helicobacter pylori, cytomegalovirus (CMV), and herpes simplex virus (HSV) has been implicated in the pathogenesis of atherosclerosis. The carotid intima-media thickness (IMT) can be taken to indicate early atherosclerosis, the presence of a carotid stenosis is a marker of a manifest carotid atherosclerosis, and an increase in arterial stiffness is used as marker of structural and functional changes in an atherosclerotic vessel wall.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.
Having issues? You can contact us here
Accelerating Research

Address

John Eccles House
Robert Robinson Avenue,
Oxford Science Park, Oxford
OX4 4GP, United Kingdom