Are Morphological or Functional Changes in the Carotid Artery Wall Associated With Chlamydia pneumoniae, Helicobacter pylori, Cytomegalovirus, or Herpes Simplex Virus Infection?
Author(s) -
Christine EspinolaKlein,
HansJürgen Rupprecht,
Stefan Blankenberg,
Christoph Bickel,
Helmuth Kopp,
Gerd Rippin,
Gerd Häfner,
Ulf Pfeifer,
Jürgen Meyer
Publication year - 2000
Publication title -
stroke
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 3.397
H-Index - 319
eISSN - 1524-4628
pISSN - 0039-2499
DOI - 10.1161/01.str.31.9.2127
Subject(s) - medicine , chlamydia , helicobacter pylori , herpes simplex virus , intima media thickness , stenosis , cytomegalovirus , pathogenesis , gastroenterology , chlamydophila pneumoniae , cardiology , serology , immunology , antibody , virus , carotid arteries , herpesviridae , chlamydiaceae , viral disease
Chronic infection with Chlamydia pneumoniae, Helicobacter pylori, cytomegalovirus (CMV), and herpes simplex virus (HSV) has been implicated in the pathogenesis of atherosclerosis. The carotid intima-media thickness (IMT) can be taken to indicate early atherosclerosis, the presence of a carotid stenosis is a marker of a manifest carotid atherosclerosis, and an increase in arterial stiffness is used as marker of structural and functional changes in an atherosclerotic vessel wall.
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