Effects of S -Nitrosoglutathione on Acute Vasoconstriction and Glutamate Release After Subarachnoid Hemorrhage | Zendy

Fatima A. Sehba | Zendy; Wan Hong Ding | Zendy; И. А. Черешнев | Zendy; Joshua B. Bederson | Zendy

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Effects of S -Nitrosoglutathione on Acute Vasoconstriction and Glutamate Release After Subarachnoid Hemorrhage

Author(s) -

Fatima A. Sehba,

Wan Hong Ding,

И. А. Черешнев,

Joshua B. Bederson

Publication year - 1999

Publication title -

stroke

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.397

H-Index - 319

eISSN - 1524-4628

pISSN - 0039-2499

DOI - 10.1161/01.str.30.9.1955

Subject(s) - medicine , vasoconstriction , subarachnoid hemorrhage , anesthesia , cerebral blood flow , intracranial pressure , saline , vasospasm , mean arterial pressure , cerebral perfusion pressure , blood pressure , heart rate

Subarachnoid hemorrhage (SAH) causes acute vasoconstriction that contributes to ischemic brain injury shortly after the initial bleed. It has been theorized that decreased availability of nitric oxide (NO) may contribute to acute vasoconstriction. Therefore we examined the effect of the NO donor N-nitroso glutathione (GSNO) on acute vasoconstriction and early ischemic glutamate release after experimental SAH.

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