Increased Platelet Activation in the Chronic Phase After Cerebral Ischemia and Intracerebral Hemorrhage | Zendy

Fop van Kooten | Zendy; Giovanni Ciabattoni | Zendy; Peter J. Koudstaal | Zendy; Diederik W.J. Dippel | Zendy; Carlo Patrono | Zendy

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Increased Platelet Activation in the Chronic Phase After Cerebral Ischemia and Intracerebral Hemorrhage

Author(s) -

Fop van Kooten,

Giovanni Ciabattoni,

Peter J. Koudstaal,

Diederik W.J. Dippel,

Carlo Patrono

Publication year - 1999

Publication title -

stroke

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.397

H-Index - 319

eISSN - 1524-4628

pISSN - 0039-2499

DOI - 10.1161/01.str.30.3.546

Subject(s) - medicine , intracerebral hemorrhage , stroke (engine) , aspirin , excretion , platelet activation , urinary system , creatinine , gastroenterology , platelet , atrial fibrillation , thromboxane , endocrinology , cardiology , anesthesia , subarachnoid hemorrhage , mechanical engineering , engineering

Enhanced thromboxane (TX) biosynthesis has previously been reported in the acute phase after ischemic stroke. We investigated whether enhanced urinary excretion of 11-dehydro-TXB2, a noninvasive index of platelet activation, was present in the chronic phase after a transient ischemic attack (TIA) or stroke, including intracerebral hemorrhage.

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