Potentiation by hypoxia of contractions caused by angiotensin II in dog and monkey cerebral arteries. | Zendy

Kazushige Yoshida | Zendy; Tomonori Okamura | Zendy; Noboru Toda | Zendy

AI Assistant Blog Pricing

Home ZAIA Blog

Open Access

Potentiation by hypoxia of contractions caused by angiotensin II in dog and monkey cerebral arteries.

Author(s) -

Kazushige Yoshida,

Tomonori Okamura,

Noboru Toda

Publication year - 1993

Publication title -

stroke

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.397

H-Index - 319

eISSN - 1524-4628

pISSN - 0039-2499

DOI - 10.1161/01.str.24.3.421

Subject(s) - hypoxia (environmental) , medicine , endocrinology , prostaglandin , angiotensin ii , contraction (grammar) , arachidonic acid , long term potentiation , cerebral arteries , renin–angiotensin system , prostaglandin e2 , prostaglandin e , vasoconstriction , blood pressure , chemistry , biochemistry , oxygen , receptor , organic chemistry , enzyme

Hypoxia alters the responsiveness to endogenous substances of cerebral arteries, possibly resulting in the modulation of blood supply to ischemic brain regions. The present study was undertaken to analyze the mechanism of potentiation by hypoxia of angiotensin II-induced cerebroarterial contractions.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research