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Large transmural myocardial infarction (MI) leads to maladaptive cardiac remodeling and places patients at increased risk of congestive heart failure. Angiotensin II, endothelin, and alpha1-adrenergic receptor agonists are implicated in the development of cardiac hypertrophy, interstitial fibrosis, and heart failure after MI. Because these agonists are coupled to and activate Gq alpha protein in the heart, the aim of the present study was to investigate Gq alpha expression and function in cardiac remodeling and heart failure after MI.

Expression of G qα and PLC-β in Scar and Border Tissue in Heart Failure Due to Myocardial Infarction

Expression of G _qα and PLC-β in Scar and Border Tissue in Heart Failure Due to Myocardial Infarction