Cellular mechanisms of captopril-induced matrix remodeling in Syrian hamster cardiomyopathy. | Zendy

Glenn Davison | Zendy; Christopher S. Hall | Zendy; James G. Miller | Zendy; M.J. Scott | Zendy; Samuel A. Wickline | Zendy

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Cellular mechanisms of captopril-induced matrix remodeling in Syrian hamster cardiomyopathy.

Author(s) -

Glenn Davison,

Christopher S. Hall,

James G. Miller,

M.J. Scott,

Samuel A. Wickline

Publication year - 1994

Publication title -

circulation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 7.795

H-Index - 607

eISSN - 1524-4539

pISSN - 0009-7322

DOI - 10.1161/01.cir.90.3.1334

Subject(s) - captopril , hamster , medicine , heart failure , myocyte , endocrinology , angiotensin converting enzyme , cardiomyopathy , cardiology , blood pressure

Although angiotensin-converting enzyme (ACE) inhibitors have become a mainstay of treatment for chronic congestive heart failure (CHF), it is not known whether the cardiac remodeling effects are a secondary phenomenon, resulting from ACE inhibitors' hemodynamic actions of afterload reduction, or occur through an independent mechanism.

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