Site of intimal rupture or erosion of thrombosed coronary atherosclerotic plaques is characterized by an inflammatory process irrespective of the dominant plaque morphology. | Zendy

Allard C. van der Wal | Zendy; A E Becker | Zendy; Chris M. van der Loos | Zendy; P. K. Das | Zendy

Open Access

Site of intimal rupture or erosion of thrombosed coronary atherosclerotic plaques is characterized by an inflammatory process irrespective of the dominant plaque morphology.

Author(s) -

Allard C. van der Wal,

A E Becker,

Chris M. van der Loos,

P. K. Das

Publication year - 1994

Publication title -

circulation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 7.795

H-Index - 607

eISSN - 1524-4539

pISSN - 0009-7322

DOI - 10.1161/01.cir.89.1.36

Subject(s) - fibrous cap , medicine , atheroma , thrombosis , inflammation , lesion , myocardial infarction , pathology , artery , macrophage , foam cell , vulnerable plaque , acute coronary syndrome , coronary thrombosis , monoclonal antibody , antibody , cardiology , cholesterol , immunology , in vitro , lipoprotein , biology , biochemistry

The study was designed to verify the concept of plaques "at risk" and whether inflammation could play a role in plaque rupture and thrombosis.

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