Relation of plasma lipoprotein(a) to infarct artery patency in survivors of myocardial infarction.
Author(s) -
David J. Moliterno,
Richard A. Lange,
R S Meidell,
J. E. Willard,
C C Leffert,
Robert D. Gerard,
Eric Boerwinkle,
Helen H. Hobbs,
L.David Hillis
Publication year - 1993
Publication title -
circulation
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 7.795
H-Index - 607
eISSN - 1524-4539
pISSN - 0009-7322
DOI - 10.1161/01.cir.88.3.935
Subject(s) - medicine , myocardial infarction , cardiology , fibrinolysis , thrombolysis , fibrinogen , timi , tissue plasminogen activator , infarction , coronary artery disease , artery , lipoprotein(a) , thrombus , plasminogen activator , lipoprotein , cholesterol
In the minutes to days after myocardial infarction, endogenous lysis of an occlusive coronary arterial thrombus occurs in most subjects. Compared with those in whom thrombolysis does not occur, those with antegrade flow in the infarct artery have improved left ventricular performance, less left ventricular dilatation, and improved survival. This study was performed to assess intrinsic hemostasis and fibrinolysis in survivors of myocardial infarction with or without antegrade perfusion of the infarct artery.
Accelerating Research
Robert Robinson Avenue,
Oxford Science Park, Oxford
OX4 4GP, United Kingdom
Address
John Eccles HouseRobert Robinson Avenue,
Oxford Science Park, Oxford
OX4 4GP, United Kingdom