Significance of smoking in patients receiving thrombolytic therapy for acute myocardial infarction. Experience gleaned from the International Tissue Plasminogen Activator/Streptokinase Mortality Trial.
Author(s) -
Gabriel I. Barbash,
Harvey D. White,
Michaela Modan,
Rafael Díaz,
J R Hampton,
J Heikkilä,
Arni Kristinsson,
S Moulopoulos,
Ernesto Paolasso,
T Van der Werf
Publication year - 1993
Publication title -
circulation
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 7.795
H-Index - 607
eISSN - 1524-4539
pISSN - 0009-7322
DOI - 10.1161/01.cir.87.1.53
Subject(s) - medicine , streptokinase , killip class , myocardial infarction , tissue plasminogen activator , diabetes mellitus , univariate analysis , stroke (engine) , surgery , cardiology , multivariate analysis , percutaneous coronary intervention , endocrinology , mechanical engineering , engineering
Despite the fact that smoking is a well-established risk factor for the development of coronary artery disease, some investigators have noted that hospital mortality after acute myocardial infarction is lower in patients who smoke than in nonsmoking patients. To evaluate the association of smoking with mortality during hospitalization after thrombolytic therapy and 6 months afterward, we analyzed the results of the International Tissue Plasminogen Activator/Streptokinase Mortality Trial.
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