Myocardial adrenergic denervation supersensitivity depends on a postreceptor mechanism not linked with increased cAMP production. | Zendy

H. Kirk Hammond | Zendy; David A. Roth | Zendy; Charles E. Ford | Zendy; G W Stamnas | Zendy; Michael G. Ziegler | Zendy; CN Ennis | Zendy

Open Access

Myocardial adrenergic denervation supersensitivity depends on a postreceptor mechanism not linked with increased cAMP production.

Author(s) -

H. Kirk Hammond,

David A. Roth,

Charles E. Ford,

G W Stamnas,

Michael G. Ziegler,

CN Ennis

Publication year - 1992

Publication title -

circulation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 7.795

H-Index - 607

eISSN - 1524-4539

pISSN - 0009-7322

DOI - 10.1161/01.cir.85.2.666

Subject(s) - denervation , medicine , endocrinology , adenylyl cyclase , stimulation , adrenergic , adrenergic receptor , catecholamine , receptor

Two major hypotheses have been proposed to explain catecholamine supersensitivity after myocardial denervation, but neither sufficiently explains certain features of the phenomenon. In addition, a nonsurgical method for long-term myocardial adrenergic denervation is desirable but has not been accomplished or described with respect to catecholamine supersensitivity.

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