Response of high-energy phosphates and lactate release during prolonged regional ischemia in vivo. | Zendy

Saul Schaefer | Zendy; Gregory G. Schwartz | Zendy; J A Wisneski | Zendy; Steven D. Trocha | Zendy; I Christoph | Zendy; S. Steinman | Zendy; Javier O. Garcia | Zendy; Barry M. Massie | Zendy; Michael W. Weiner | Zendy

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Response of high-energy phosphates and lactate release during prolonged regional ischemia in vivo.

Author(s) -

Saul Schaefer,

Gregory G. Schwartz,

J A Wisneski,

Steven D. Trocha,

I Christoph,

S. Steinman,

Javier O. Garcia,

Barry M. Massie,

Michael W. Weiner

Publication year - 1992

Publication title -

circulation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 7.795

H-Index - 607

eISSN - 1524-4539

pISSN - 0009-7322

DOI - 10.1161/01.cir.85.1.342

Subject(s) - phosphocreatine , ischemia , medicine , intracellular ph , acidosis , high energy phosphate , hibernating myocardium , in vivo , endocrinology , blood flow , adenine nucleotide , metabolism , energy metabolism , anesthesia , intracellular , biochemistry , chemistry , biology , revascularization , myocardial infarction , nucleotide , microbiology and biotechnology , gene

The functional impairment of persistently ischemic, or "hibernating," myocardium may serve to maintain myocardial cell viability through a reduction of energy requirements. Although previous studies have, in a variety of experimental models, independently shown variable responses in lactate metabolism and intracellular phosphates during prolonged ischemia, the responses of these metabolites under identical flow conditions have not been adequately described.

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