Dobutamine increases cardiac output of the total artificial heart. Implications for vascular contribution of inotropic agents to augmented ventricular function. | Zendy

Philip F. Binkley | Zendy; Kevin D. Murray | Zendy; Kim M. Watson | Zendy; P. David Myerowitz | Zendy; Carl V. Leier | Zendy

Open Access

Dobutamine increases cardiac output of the total artificial heart. Implications for vascular contribution of inotropic agents to augmented ventricular function.

Author(s) -

Philip F. Binkley,

Kevin D. Murray,

Kim M. Watson,

P. David Myerowitz,

Carl V. Leier

Publication year - 1991

Publication title -

circulation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 7.795

H-Index - 607

eISSN - 1524-4539

pISSN - 0009-7322

DOI - 10.1161/01.cir.84.3.1210

Subject(s) - dobutamine , medicine , inotrope , cardiology , vascular resistance , cardiac output , heart failure , contractility , hemodynamics , stroke volume , anesthesia , ejection fraction

The synthetic catecholamine dobutamine increases stroke volume in normal subjects and in patients with congestive heart failure. In addition to its direct influence on myocardial contractility, dobutamine may significantly modulate vascular tone because of its alpha- and beta-adrenergic agonist activity.

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