SLC1A5 Silencing Inhibits Esophageal Cancer Growth via Cell Cycle Arrest and Apoptosis | Zendy

Jieping Lin | Zendy; Teng Yang | Zendy; Peng Zheng | Zendy; Haiyan Xiao | Zendy; Na Jiang | Zendy; Lifang Zhang | Zendy; Dickerson CA | Zendy; Ping Wu | Zendy; Qingjun Pan | Zendy

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SLC1A5 Silencing Inhibits Esophageal Cancer Growth via Cell Cycle Arrest and Apoptosis

Author(s) -

Jieping Lin,

Teng Yang,

Peng Zheng,

Haiyan Xiao,

Na Jiang,

Lifang Zhang,

Dickerson CA,

Ping Wu,

Qingjun Pan

Publication year - 2018

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000491769

Subject(s) - gene knockdown , glutamine , apoptosis , transfection , cancer research , programmed cell death , cancer cell , esophageal cancer , cancer , cell growth , cell cycle , chemistry , biology , cell culture , amino acid , biochemistry , genetics

Our data indicate that SLC1A5 plays an important role in esophageal cancer both in vivo and in vitro. The inhibition of esophageal cancer growth by targeting SLC1A5 could, therefore, be used as a preoperative therapy for esophageal cancer.

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