Camel Milk Ameliorates 5-Fluorouracil-Induced Renal Injury in Rats: Targeting MAPKs, NF-κB and PI3K/Akt/eNOS Pathways | Zendy

Hany H. Arab | Zendy; Samir A. Salama | Zendy; Ibrahim A. Maghrabi | Zendy

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Camel Milk Ameliorates 5-Fluorouracil-Induced Renal Injury in Rats: Targeting MAPKs, NF-κB and PI3K/Akt/eNOS Pathways

Author(s) -

Hany H. Arab,

Samir A. Salama,

Ibrahim A. Maghrabi

Publication year - 2018

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000489210

Subject(s) - enos , protein kinase b , oxidative stress , proinflammatory cytokine , pi3k/akt/mtor pathway , nephrotoxicity , chemistry , apoptosis , mapk/erk pathway , p38 mitogen activated protein kinases , pharmacology , inflammation , cancer research , kidney , endocrinology , medicine , signal transduction , nitric oxide synthase , nitric oxide , biochemistry

The clinical utility of 5-fluorouracil (5-FU) is limited by its nephrotoxicity. Camel milk (CM) has previously displayed beneficial effects in toxicant-induced nephropathies. The current study aimed to investigate the potential of CM to attenuate 5-FU-induced nephrotoxicity in rats.

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