Crocin Improves the Endothelial Function Regulated by Kca3.1 Through ERK and Akt Signaling Pathways | Zendy

Huike Yang | Zendy; Xuemei Li | Zendy; Yang Liu | Zendy; Xinlei Li | Zendy; Xiaodong Li | Zendy; Mengnan Wu | Zendy; Xiaohong Lv | Zendy; Chun Chunhua | Zendy; Xuansheng Ding | Zendy; Yafang Zhang | Zendy

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Crocin Improves the Endothelial Function Regulated by Kca3.1 Through ERK and Akt Signaling Pathways

Author(s) -

Huike Yang,

Xuemei Li,

Yang Liu,

Xinlei Li,

Xiaodong Li,

Mengnan Wu,

Xiaohong Lv,

Chun Chunhua,

Xuansheng Ding,

Yafang Zhang

Publication year - 2018

Publication title -

cellular physiology and biochemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.486

H-Index - 87

eISSN - 1421-9778

pISSN - 1015-8987

DOI - 10.1159/000488735

Subject(s) - crocin , enos , protein kinase b , mapk/erk pathway , pharmacology , chemistry , thrombomodulin , viability assay , nitric oxide , microbiology and biotechnology , endothelium , nitric oxide synthase , signal transduction , medicine , biology , biochemistry , immunology , thrombin , cell , platelet , organic chemistry

Based on the protective effect of crocin against cardiovascular diseases, we hypothesize that crocin could improve endothelial function through activating the eNOS(endothelial nitric oxide synthase) /NO pathway and/or the intermediate-conductance Ca2+-activated K+ channels (KCa3.1).

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